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Sleepwalking 'linked to chromosome fault'

2011-02-08 10:46:15

By Michelle Roberts Health reporter, BBC News

Scientists believe they have discovered the genetic code that makes some people

sleepwalk.

By studying four generations of a family of sleepwalkers they traced the fault

to a section of chromosome 20.

Carrying even one copy of the defective DNA is enough to cause sleepwalking,

the experts told the journal Neurology.

They hope to target the genes involved and find new treatments for the

condition that affects up to 10% of children and one in 50 adults.

Most often, sleepwalking is a fairly benign problem and something that will be

outgrown.

Start Quote

What we have found is the first genetic locus for sleepwalking

End Quote Dr Christina Gurnett Lead researcher

Many children will have episodes where they will arise from their sleep in a

trance-like state and wander.

But more extreme cases of sleepwalking can be deeply disruptive and downright

dangerous, particularly when the condition persists into adulthood.

Stress trigger

Sleepwalkers may perform complex feats such as locating the car keys, unlocking

the doors and then driving.

There have even been high-profile cases where sleepwalkers have killed during

an episode.

Despite this relatively little is known about the phenomenon, called

somnambulism by medics.

Sleepwalking

Moon

has fallen asleep

getting dressed and leaving the house

bed of their own accord

Experts do know that sleepwalking tends to run in families and that some people

are particularly susceptible to it.

And factors like being over-tired or stressed can be the trigger.

Typically, episodes happen early in the night, soon after the individual has

fallen asleep and is in the deep, dreamless "slow wave" or non-rapid eye

movement (NREM) stage of sleep.

By morning, the person will usually have no recollection of the episode.

For the latest study, Dr Christina Gurnett and colleagues at the Washington

University School of Medicine sought the help of a large family of

sleepwalkers.

The family had been referred to them because one of the youngest members, a

12-year-old girl called Hannah, had been experiencing particularly troublesome

sleepwalking, which regularly caused her to leave the house and roam during the

night.

Among the four generations of the family, spanning from the great-grandparents

downwards, nine members out of the 22 were sleepwalkers.

One family member - an uncle of Hannah's - frequently wakes to find he has put

on eight pairs of socks during the night. Some of her other sleepwalking

relatives have suffered injuries such as broken toes during their nocturnal

wanderings.

Child's feet Sleepwalking is more common during childhood

Using saliva samples the researchers analysed the family's DNA to unpick the

genetics of the condition.

A genome-wide search revealed the problem stemmed from genetic code housed on

chromosome 20, and that this code had been passed down from generation to

generation. Someone with the gene has a 50% chance of passing it on to their

children.

And any individual who inherited a copy of the faulty DNA would be a

sleepwalker, they found.

Although they have yet to identify the precise gene or genes involved - there

are a potential 28 - their hunch is that it will be the adenosine deaminase

gene that is the culprit.

This gene, which sits in the minute segment of chromosome 20 that the

researchers identified, is already known to be linked to the slow wave sleep

that sleepwalking occurs within.

Dr Gurnett said: "It is likely that several genes will be involved. What we

have found is the first genetic locus for sleepwalking.

"We do not know yet which of the genes in this linkage region of chromosome 20

will be responsible. Until we find the gene we won't know whether this accounts

for several families or a large number of families who have sleepwalking.

"But discovering these genes could help with identifying and treating the

condition."

Dr Malcolm von Schantz, a sleep expert at the University of Surrey, said: "This

provides the proof of concept. We are beyond the needle in the haystack stage.

It's now become feasible to find out which mutation in which gene is

responsible."